Share The Wealth by Chris Gupta
May 12, 2008

Curing the Incurable: Vitamin C, Infectious Diseases, & Toxins

Dr. Levy, MD has written much on Vitamin C. His book "Curing the Incurable: Vitamin C, Infectious Diseases, & Toxins" is an absolute gold mine.You can read parts of first 5 chapters of the book here. Chock full of references (over 35 pages)! This alone is worth the price of the book many times over. Here we have an absolute abundance of peer reviewed literature that no drug can match - and the looney bin faction of the medical fraternity continue to ask for evidence on the efficacy let alone use it? All researchers, MDs, health professionals and/or anyone interested in health worth their salt must read this book. It will allay any doubts once and for all on the value of Vitamin C and for that matter on Orthomolecular solutions to disease! Although I hasten to add that while Vitamin C is essential it is not, like any single nutrient, it is not sufficient on its own. Other nutrient deficiencies and body toxicities from water, food, environment and last but not least drugs must also be addressed.

In view of the propensity to spread miss information by the media on the behest of the self serving medical Mafia, who rather force down our throats toxic drugs pretending as if we have deficiency of drugs instead of nutrients. While toxicity seems to be a non issue given the drugs they foist on us, here is very important news that should preempt much nonsense regarding the lack of Vitamin C's antioxidant properties.

Remember we really do have a free media - free in that when they own it they are free to suppress anything they wish....

Chris Gupta


Vitamin C: Always an Antioxidant

At long last, intravenous vitamin C is being used with substantial frequency by many different doctors, with consistently strongly positive results. This newsletter was written to help clarify some erroneous concepts about such infusions that are being passed along now as some sort of accepted scientific fact.

The last thing this extremely useful therapeutic modality needs is to be misunderstood and no longer used appropriately.

Best regards,

Thomas E. Levy, MD, JD

Issue No. 10

March, 2008

Vitamin C: Always an Antioxidant

Vitamin C, containing the ascorbate anion in all of its various forms (ascorbic acid, sodium ascorbate, calcium ascorbate, or mineral ascorbates), is solely an antioxidant, or reducing agent. That is to say, vitamin C only has electrons to donate. It cannot, under any circumstances, take electrons away from surrounding tissues and act directly as an oxidizing agent. To think otherwise is to create a form of biochemistry that violates the natural laws of electron flow and exchange. Only oxidized vitamin C (dehydroascorbate) is capable of receiving electrons, and this is never the type of vitamin C that is administered as a supplement or a therapy.

There appears to have developed a pattern of thinking that high-dose vitamin C therapy can somehow change the basic chemical characteristics of vitamin C, such that vitamin C becomes an oxidizing agent and stops acting as a reducing agent at elevated doses. In other words, an increasing number of individuals are asserting that vitamin C will somehow assume a different chemical nature above a certain dose and start taking electrons out of its surrounding environment rather than continuing to donate them. Some individuals are even asserting that high-dose vitamin C is always prooxidant, an outlandishly incorrect notion. This is very flawed thinking, which can ultimately lead to clinical decisions that are not in the best interests of the patients being treated.

What mayCuring the Incurable: Vitamin C, Infectious Diseases, & Toxins have caused this continuing confusion over the nature of vitamin C is that there are at least two circumstances under which the administration of vitamin C (or any other antioxidant) can result in a clinically definable net prooxidant effect. In other words, under such conditions the administration of vitamin C would appear to be oxidizing its surroundings rather than reducing them. However, it is extremely important to emphasize that it is a overall clinical effect of apparent oxidation that is taking place, and not that vitamin C has suddenly stopped donating electrons and started taking them away. This is a very important point to understand, and it is of much more than just academic interest that this point is appreciated.

In one well-documented situation, a defined path of electron transfer known as the Fenton reaction occurs. This usually only occurs to the point that it is noticeable clinically when a lower amount of vitamin C is present with a larger than normal presence of ferric (Fe3+) or cupric (Cu2+) ion [iron or copper]. In the presence of these ions at just the right limited dose of vitamin C, the ascorbate entity donates its electrons and reduces these ions to their ferrous (Fe2+) or cuprous (Cu1+) forms. These reduced metal ions are then able to pass these donated electrons along to any hydrogen peroxide locally present and thereby generate the hydroxyl radical, an extremely reactive and oxidizing free radical. Hydrogen peroxide is normally poorly reactive but the contribution of electrons to it result in a prompt oxidative effect. The hydroxyl radical produced by this electron donation to hydrogen peroxide will oxidize (take electrons away from) virtually any organic molecule to which it is exposed. However, larger amounts of vitamin C will reliably overwhelm this typically limited and very focal generation of hydroxyl radical, promptly repairing any oxidative damage done by it and keeping the observable net clinical effect to one of antioxidation and not oxidation.

It should be kept in mind that the Fenton reaction, with the ability to very focally cause a profound oxidative effect where the proper concentrations of iron or copper are situated in the presence of hydrogen peroxide and vitamin C, represents one of the important mechanisms in which appropriately dosed vitamin C can have a cancer cell-killing effect. Iron concentrations inside some cancer cells make them especially susceptible to properly dosed vitamin C by the intracellular generation of free radicals such as the hydroxyl radical noted above. Indeed, if such mechanisms did not exist, vitamin C would strongly support the health of everything in the body, including microorganisms and cancer cells. However, quite the opposite is true, and such mechanisms as the Fenton reaction need to exist for vitamin C to have its well-documented ability to target many different infections and some types of cancer cells, while strongly supporting the health of the normal cells and tissues in the body.

In another clinical situation, much less well-documented in the scientific literature than the Fenton reaction, a given individual will react in what clinically appears to be an unpredictable and idiosyncratic manner. When such an individual receives a larger, multigram dose of vitamin C intravenously, there can appear some degree of clinical deterioration, rather than the much more typical response of a substantial and improved degree of clinical well-being. What is happening here is that most adults have significant intracellular stores of a wide variety of toxins and toxic metabolic wastes. Just like the body has systems of elimination, the individual cell has such systems as well. In the cell, however, the process of elimination is powered by a number of different enzyme-driven mechanisms. In the very toxic individual, much of this enzyme function is itself suppressed or neutralized by the very toxins that are needed to be processed and eliminated. In such an individual, the sudden presence of a large influx of electrons such as is provided by an intravenous infusion of vitamin C results in a rapid "reactivation" of these enzymes, and toxins end up being excreted from the cells and effectively "dumped" quite rapidly into the bloodstream. When this occurs, the patient feels poorly, or at least less well. Ironically, but logically, the cessation of the infusion for a brief period of time (30 minutes to an hour), followed by a reinitiation of the infusion at a slower rate, results in rapid clinical improvement as the circulating vitamin C then "mops up" the prooxidative toxin effects while not continuing to cause as rapid a dump of toxins into the bloodstream.

To recap, then, vitamin C can never be anything other than an electron-donating antioxidant. However, in at least the two circumstances described above, the net effect of the vitamin C can result in a clinical picture of prooxidation and increased symptoms of toxicity. However, these occurrences are relatively rare, and when they do occur they are easily remedied when the mechanisms behind them are understood.

Copyright: 2008 by Thomas E. Levy, M.D., J.D.
All Rights Reserved; Reproduction Permitted only with Acknowledgement and of the Entire Document

Consider forwarding this newsletter to your personal email lists or to specific friends who may be interested in the information. Thanks

See also:

Conquering Cancer Through Vitamin C & Other Antioxidants

The 1982 Letter On Vitamin C and Cancer

Excessive Vitamin C Consumption Does Not Cause Kidney Stones!

Lyme Disease -- Wiped Out By Vitamin C

The Stonewalling of Vitamin C

Vitamin C: Good for G

All Vitamin C/Common Cold Studies Conducted Over The Past 60 Years Are Flawedout

How to Get Intravenous Vitamin C Given to a Hospitalized Patient


The Negative Impact of Sugar on Vitamin C



Vitamin C & Menopause

Vitamin C and SARS


posted by Chris Gupta on Monday May 12 2008

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