Cancer: The ATP-Photon Hypothesis

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Health

Could cancer be a result of the functional degradation of a cellular photon absorption pathway that is basic to the production of ATP, the cellular energy molecule? And could tumor cells merely be executing an age-old survival program they retained from the time of unicellular existence? An interesting new hypothesis of cancer causation spells out why some cells all of a sudden start dividing to form tumors, and how that urge to sustained growth could likely be 'switched off. Heinrich Kremer MD, a German doctor known for his dissident work in the area of AIDS, has made a revolutionary discovery on cancer genesis, based on research into cell symbiosis.

Kremer points out that in cancer, there is a functional breakdown of a photon-mediated pathway for ATP synthesis in the mitochondria of our cells. This eventually leads to what might be called a personality change of cells. Losing communication with each other and thus believing that they are 'on their own', the cancer cells revert back to a prehistoric state where the only way to survive was to multiply and form colonies. These colonies form tumors in the human organism and at times cause problems, such as reduced circulation or impaired function of an organ.

Kremer also maintains that our current understanding of how cells produce the energy molecule ATP is fundamentally flawed, and he says that is why our efforts to find a cure for cancer have so far failed.

ATP production, according to Kremer's research, is not based on chemical energy release, as taught in universities today, but rests on the absorption of photons of light from the zero-point quantum medium. The details of Kremer's discovery are absolutely fascinating and the new theory also points to a simple biological cure. Curcuma, a root vegetable used abundantly in oriental cooking, has anti-cancer properties and Kremer explains how it is able to intervene in the process to turn the tide on the disease.

Turmeric (Curcuma longa) - curative spice and coloring agent - Image credit: Wilsonart

Dr Kremer's paradigm changing discovery brings to mind another piece of cancer research that made headlines recently: Researchers at the University of Alberta have found that dichloroacetate (DCA), a simple, cheaply available substance, can successfully revive the mitochondria - our cellular ATP generators - which are somehow switched off in cancer cells. That Canadian research is being kept quiet though, because no one has figured out yet how or why DCA works. Perhaps Kremer's discovery of the ATP photon pathway could shed some light?

Kremer's article was first published in the Townsend Letter for Doctors and Patients - August/September 2007. Here is a copy:

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The secret of cancer: “short-circuit” in the photon switch

by Heinrich Kremer, MD

(original here)

Change in the medical world-view of tumorology - The rational Cell Symbiosis Therapy concept


In western countries every third person suffers from some form of cancer, every fourth person dies of it. The prognoses of the WHO state that by the year 2050 half of all mortalities will be due to a cancerous disease. According to the prevailing cancer theories chance defects (mutations) in the DNA in the nucleus, which are regarded as irreparable, are considered to be the primary cause of the disease. Standard therapy in oncology (operations, chemotherapy and/or radiation therapy) is based on this assumption. The cure rates of cancer (minimum of 5 years survival after diagnosis) are given as being 45% (22% surgical treatment, 12% radiation therapy, 5% chemotherapy, 6% combined standard therapies). 60 –70% of patients with incurable cancer are palliatively treated with radiation therapy, 50% with chemotherapy and less than 1% of the patients are treated surgically (EU data, 2003). In the USA, for instance 20% of the overall health budget is spent annually on chemotherapy for cancer patients.

The Nobel Prize winner Professor Watson, who together with Crick discovered the double helix of DNA in the nucleus, the most prominent promoter of the 1971 “War on Cancer” succinctly declared in 2003: “First we have to understand cancer before we can cure it”. The background to this sobering thought after decades of most intensive research efforts and a massive capital injection is the fact that the classic mutation theory of oncogenesis has been forever shaken by newer research. Under the mutation theory a tumor colony develops from a single “degenerated” body cell that through uncontrolled division is thought to pass on identical DNA defects to all daughter cells. However, it has become apparent that each individual cancer cell, even within the same tumor of a patient, features a different genetic variation.

The internationally respected cancer researchers Professor Weinberg from the MIT in Cambridge, USA and Professor Hahn from the Dana Farber Cancer Research Center in Boston, both supporters of the classic mutation theory, published in 2002 an overview of the ostensibly still puzzling six “diabolic acquired capabilities” of cancer cells. These attributes include the ability to:

1. resist exogenous growth-inhibitory signals
2. generate their own mitogenic signals
3. bypass apoptosis
4. acquire vasculature
5. gain potential immortality
6. invade and metastasize

The “Cell Dyssmybiosis Concept” (Kremer 2001) explained for the first time the six “acquired capabilities” of cancer cells as an evolutionary-biologically programmed natural (albeit overregulated) protective switch of the divisionally active human cells during permanent chronic cell stress. The origin of this concept was the evolutionary-biological discovery that humans owe their biological existence, like all nucleic single- or multi-cellular creatures (eukaryotes), to a unique act of integration deep in the history of evolution. Roughly 2 billion years ago two unicellular organisms without nuclei from the archaea and bacteria domains fused to a new single cell type that is now termed protista. Comprehensive comparative sequence analyses regarding the genetic make-up and specific proteins of archaea, bacteria and a multitude of eukaryotic organisms including humans produced an astonishing result: About 60% of the genes in a human nucleus originate from the primeval archaea (A genome) the remaining genes having a bacterial origin (B genome), which in particular in the nucleus are delegated by the bacterial symbionts that have survived up until today in all human cells as mitochondria (on average 1,500 per cell).

There is a controlled division of labour between the A genome and the B genome: The A genome dominates the late cell division phases, the B genome drives the early cell division phase and the differentiated cell performances of the respective cell types. From these fundamental cellular biological facts, on the basis of the integration of a large number of new experimental and clinical research data the cell symbiosis concept leads to the following conclusions about oncogenesis and cancer therapy:

1. There is a controlled alternate switch between the mitochondria and both nuclear subgenomes

2. Transformation to cancer cells is a functional (not structural) failure of this alternate switch, after the divisional phase cells are no longer sufficiently able to switch back to the differentiated cell performance phases

3. The cause of this permanent function failure is the gradual deficiency of one of the central functions of mitochondria, namely to supply ca. 90% of the “universal energy-storing and energy-transporting molecule” adenosine triphosphate (ATP) for practically all biosyntheses and metabolic processes. Under normal circumstances roughly one’s body weight of ATP has to be synthesized and then broken up every day. ATP cannot be stored and the actual stock in human beings is enough for only 5 seconds. When the mitochondrial functions are disturbed cancer cells switch intermittently or permanently to the archaic form of ATP synthesis in the cytoplasm (glycolysis) with, potentially, up to a 20-fold increase in the glucose turnover at the cost of the organism as a whole (cachexia resulting from the forced degradation, especially of muscle proteins for the benefit of carbon intermediary products for glycolysis, is one of the most frequent causes of death in cancer patients)

4. Hitherto perceptions about the synthesis and function of ATP molecules, the basis of all cellular biological medical theories, are, however, objectively false. ATP has 3 molecule groups: 1 base adenine ring molecule that absorbs the light quanta near to ultra-violet levels of 270 nm, 1 sugar molecule with 5 carbon atoms as well as a 1 molecule string with 3 phosphate groups.
The current dogma, based on a theory formed more than 60 years ago by the later Nobel Prize winner Lippmann, is that electron energy is transferred in the respiratory chains of mitochondria (of which there are literally thousands in every mitochondrion as shown by EM photographs) on discharge of “energy-rich” electrons from nutrients via a kind of electrochemical battery, to protons which for their part drive ATP synthesis energetically and store their surplus energy in the phosphate bonds of ATP. These “energy-rich” phosphate bonds of ATP transported into the cytoplasm then release this stored energy via hydrolysis mainly to maintain the energetic processes of cell metabolism. Biochemical experiments have clearly shown, however, that the phosphate bonds of ATP are not especially rich in energy and on hydrolysis only heat energy is released that can at the most be used for heat production by isotherm cells (constant cell temperature). The fundamental question of the actual mechanism for the acquisition of cell energy remains unanswered. This fact explains the predominant failure of cancer prevention and therapy up until now.

5. Biochemistry and medical science have failed to this day to explain the function of the adenine groups of ATP as no biochemical reaction with this adenine ring molecule is shown. However, an understanding can be gained, within the framework of the cell symbiosis concept, from the biophysical attributes of light absorption of the adenine group. All essential components of mitochondrial cell respiration are light absorbing molecules with characteristic “frequency windows” of absorption maxima from nearly UV spectrum to the longer wave yellow/orange spectral range of visible light up